Conversely, knockdown of ALKBH5 impairs tumor formation in vivo by decreasing hypoxia‐induced NANOG expression and BCSC enrichment.41 It was also reported that overexpression of ALKBH5 promotes invasion and metastasis of gastric cancer by demethylating the lncRNA NEAT1. 42 Panneerdoss et al43 revealed that ALKBH5 exerts its pro‐tumorigenic role by regulating m6A levels of angiogenesis‐associated and epithelial‐mesenchymal transition transcripts. This evidence concerns the gene ALKBH5 and gastric cancer.