CXCL10/CXCR3 expression was analyzed in two experimental models of nephrotic syndrome, puromycin aminonucleoside nephropathy (PAN) and anti-nephrin antibody-induced nephropathy (ANA), both with slit diaphragm (SD) dysfunction resulting in proteinuria [41, 42]. This evidence concerns the gene CXCL10 and kidney disorder.