In abdominal aortic aneurysms (AAAs), Tsuruda et al. [40] demonstrated that GLUT-3, as well as GLUT-1, appear to be the most important isoforms mediating accumulation of 18 fluoro-deoxyglucose (18F-FDG). The gene discussed is SLC2A1; the disease is achalasia-alacrima syndrome.