To investigate whether mutations in KRAS correlate with CN-alterations, copy neutral loss of heterozygosity, or differences in gene expression, SNP6.0 and HG-U133 plus 2.0 microarrays were used to interrogate the six MM cell lines AMO1, U266, MM1.S, OPM2, JJN3, and L363, which have previously been analyzed by whole exome sequencing [26] and were included in the current amplicon sequencing approach. This evidence concerns the gene KRAS and Miyoshi myopathy.