N1 reversedthe increase in CK-MB caused by the infection, significantly reduced the number ofmyocardial inflammatory infiltrates, decreased the population of cardiac CD8+ Tcells associated with myocarditis progression in both T. cruziacute and chronic infections, and reduced the percentage of CD8+/CD49d+ Tlymphocytes in the spleen.28 Here, CD8A is linked to myocarditis.