The loss of FBP1 in basal‐like breast cancer cells causes increased glucose uptake and macromolecule biosynthesis, increased PKM2 activity, maintenance of ATP production under hypoxic condition and is an essential oncogenic event in epithelial to mesenchymal transition of cancer cells.34 In T24 BC cell line, the low expression of FBP1 was observed and higher glucose uptake compared with 5637 cell line, further indicating that T24 BC cell line resembles basal‐like BC subtype. This evidence concerns the gene FBP1 and breast cancer.