FMR1 and schizophrenia: The AM module, which was most enriched for MIA + CTRL microglia, showed enrichment for synaptic molecules, FMRP-, schizophrenia-, and ASD-implicated genes, while the MIA-IM cluster showed enrichment for genes with de novo mutations identified in human ASD and “M2” ASD prenatal genes (Fig. 2f).