The calcium/calmodulin-dependent enzyme eEF2K (eukaryotic elongation factor-2 kinase) that slows down the rate of protein translation by phosphorylating and inhibiting the function of the eEF2 (eukaryotic elongation factor-2) has been proposed to control the switch between autophagy and apoptosis in cancer cells in response to Akt inhibition [9]. The gene discussed is EEF2K; the disease is cancer.