TUG1 and hypertrophic cardiomyopathy: miR‐34a, sharing a highly conserved binding site with TUG1 (Figure 3A), regulates cardiac dysfunction in hypertrophic cardiomyopathy.22 After confirming the transfection efficiency of miR‐34a mimic and inhibitor in cardiomyocytes (Figure S1C,D), the dual‐luciferase reporter assay was performed.