Among them, we noticed the enriched and upregulated genes in the subsets of TNF and GVHD, IL-17 signaling, and TLR (e.g., FOS, IL1B, CXCLs) whereas downregulated antigen presentation-associated genes (e.g., NUSAP1, CNTNAP3B) in AA-MSCs. This evidence concerns the gene IL1B and graft versus host disease.