FASLG and idiopathic pulmonary fibrosis: Having shown [42], that IPF-media can induce resistance to cell death in normal lung myofibroblasts as in IPF-cells due to the elevated levels of sFasL in the medium, we blocked the cleavage of FasL that was initiated by MMP using MMP inhibitor batimastat (10 μM, for 24 h), compared to the vehicle (DMSO).