HNRNPA1 and gastric cancer: In addition, due to the multiple‐to‐multiple relationships between miRNAs and target genes, we could not exclude the possibility that miR‐339 might inhibited GC development and metastasis via multiple mechanisms.31 Taken together, we demonstrated that RP11‐81H3.2 could promote the GC progression through RP11‐81H3.2‐miR339‐HNRNPA1 interaction network, which provides a novel diagnosis and therapeutic marker for GC treatment.