We observed WNT/β‐catenin signalling to be active during TGFB‐induced peritoneal membrane injury, and our results suggest cross‐talk with the TGFB pathway during the development of injury.14 We used Dickkopf‐related protein (DKK‐1) to inhibit WNT/β‐catenin signalling and observed a decrease in both angiogenesis and also epithelial to mesenchymal transition (EMT).14 In a similar study of dialysis infused peritoneal fibrosis, β‐catenin was shown to induce EMT.15 Our results concur with reports of WNT signalling in other fibrotic diseases.9, 10, 11, 16, 17, 18. This evidence concerns the gene TGFB1 and Peritoneal Fibrosis.