ALDH1A1 and neoplasm: Furthermore, high AXL and p-AXL levels in the AXL-high clone strongly induced the tumourigenicity of H1299 cells in SCID/Beige mice, and oral administration of EGCG + GTE inhibited the tumour growth of H1299 cells by reducing p-AXL, SLUG and ALDH1A1 levels.