CCK8, colony formation, and transwell assays showed that cellular proliferation, migration and invasion were enhanced in cervical cancer cells with RPLP1 overexpression (Fig. 4A,C,E), and, in contrast, were inhibited in cells with RPLP1 knockdown (Fig. 4B,D,F) compared to cells without any gene modulation. The gene discussed is RPLP1; the disease is cervical cancer.