FABP1 and acute kidney injury: Increased L-FABP without creatinine-defined AKI was identified in 59% (n = 300) of patients with AKI, indicating subclinical AKI, compared with creatinine-defined AKI only, and these patients were at a greater risk of the primary endpoint than those without increased L-FABP or creatinine-defined AKI (p = 0.02).