For example, GHET1 promoted gastric carcinoma cell proliferation through increasing c‐Myc mRNA stability, and knockdown of GHET1 inhibited cell proliferation and invasion of colorectal cancer.88, 89 Guan et al found the expression of GHET1 was increased in NSCLC tissues compared with adjacent normal tissues, and knockdown of GHET1 suppressed cell proliferation and invasion. The gene discussed is MYC; the disease is non-small cell lung carcinoma.