Furthermore, in an intraperitoneal infection model, Dectin-1 knockout mice were less able to recruit neutrophils, monocytes, and eosinophils to the site of infection than wild-type mice and this was associated with a decrease in IL-6, CCL2, CCL3, G-CSF, and GM-CSF production (Taylor et al., 2007). Here, CSF3 is linked to infection.