In agreement with alternative promoters controlling UGT2B17 in PCa, no correlation between the proximal FOXA1 polymorphism located in P1 of the UGT2B17 gene and protein levels was observed in prostate tissues, in contrast to LNCaP cells.24,32 This is consistent with the predominant expression of canonical UGT2B17_v1 in LNCaP cells. The gene discussed is UGT2B17; the disease is posterior cortical atrophy.