DDR2 and neoplasm: Here we showed that the ability of COL1 scaffolds to promote tumour growth in HT1080 xenografts was only manifested in the context of ectopic DDR1b or DDR2 expression because co-implantation of COL1 with HT1080 cells in which either DDR1b or DDR2 expression was repressed (+DOX) had no impact on tumour growth (Fig. 2A,B).