Moreover, these channels were previously discovered to be somehow involved in several processes that regarded pathogenesis of endometriosis: TRPC1, TRPC4, and TRPV2 are involved in cell migration; TRPC4 in cell adhesion; and TRPM4, TRPM7, and TRPV2 have a crucial role in cell proliferation [47,61,62,63]. This evidence concerns the gene TRPC4 and endometriosis.