SLC37A2 and chronic recurrent multifocal osteomyelitis: For example, there is a gap in the reference assembly in the 5’-region of SLC37A2. Therefore, sequencing of additional genomes of well-phenotyped breed-matched obligate carriers and/or affected dogs may help unravel the complete molecular etiology of canine hyperostotic disorders such as CHS or CMO in the remaining cases by reducing the number of private candidate variants shared in cases of the same breed.