A BRG1-dependent increase in cancer cell division was assigned to (a) upregulated expression of enzymes responsible for fatty acid and lipid biosynthesis by BRG1, which binds at the loci encoding these genes; (b) induction of ABC transporter expression, particularly in response to drug treatment; and (c) association with ER and ER-mediated transcriptional activation [18,22]. Here, SMARCA4 is linked to cancer.