FCGR1A and Miyoshi myopathy: Despite in monocytes the CD64 overexpression is driven by both IL10 and IFN-γ and we have found the overexpression of ILR10B and its downstream effectors, we have not tested IL10 in vitro to recapitulate the MM-HDN phenotype since it is known that normal neutrophils do not upregulate CD64 via IL10, but only via IFN-γ13,40,41,69–71.