Conversely, CD16 was lower in both MM- and MGUS-than in healthy HDNs (respectively MFI, 945 ± 38 versus 1323 ± 82 a.u., p < 0.0001, Fig. 3D), without any significant difference of CD16 expression between MGUS and MM-HDNs. This evidence concerns the gene FCGR3A and Miyoshi myopathy.