We previously showed that 18F-FDG uptake in PPC is closely related to the presence of GLUT1 and angiogenesis, and that the accumulation of 18F-FDG and the expression level of GLUT1 were significantly higher in patients with PPC than those with other nonsmall cell lung cancer (NSCLC) [6]. This evidence concerns the gene SLC2A1 and primary peritoneal carcinoma.