In patients with Klinefelter syndrome, no association was found between biomarkers of Sertoli cell function, including AMH, and microTESE success [30]; indeed, the pattern of AMH expression in Klinefelter patients is somehow different from normal, with a delay in the FSH-mediated decline occurring in puberty, possibly due to a temporary, functional compensation of Sertoli cells [31]. Here, AMH is linked to Klinefelter syndrome.