HDAC2 and neoplasm: For example, FOXK2 inhibited the proliferation, invasion, and migration of GC cells, and its down‐regulation is related to poor prognosis in GC patients.27 Besides, HDAC2 was significantly up‐regulated in various histopathologic grades of human GC, and the inactivation of HDAC2 has been confirmed to reduce cell motility, cell invasion, clonal expansion, and tumor growth.28 Specifically, 2 TFs were found to co‐express with LINC01234 according to the co‐expression network we previously constructed.29 They are ELK1 and ZNF664 (Table S4).