A study performed on a Korean pediatric population documented the presence of a genetic mutation, namely the UNC13D mutation among children with EBV infection who developed HLH, which could partially explain their vulnerability towards exhibiting this immune disorder.[5] Due to its high prevalence in determining HLH, infection with various viral agents, including EBV needed to be searched for. This evidence concerns the gene UNC13D and Epstein-Barr virus infection.