Even further, a study in coronary thrombus aspirates from patients who had suffered an acute myocardial infarction demonstrated that up to 10–30% of the occluding thrombus mass is represented by neutrophil extracellular traps; in this study, IL-17A and IL-17F appeared to be important constituents of fresh, but not chronic, thrombi, indicating a possible role also for IL-17-dependent inflammation in acute thrombosis (113). This evidence concerns the gene IL17A and acute myocardial infarction.