Recently, Bidkhori et al19 had identified three HCC subtypes named iHCC1, iHCC2, and iHCC3 and pointed out that tumours in iHCC1 had the highest FAO fluxes, whereas 75% iHCC2 tumours carried mutations in CTNNB1, with up‐regulated expression of β‐catenin target genes (like glutamine synthetase GLUL and glutamate transporter SLC1A2). The gene discussed is CTNNB1; the disease is neoplasm.