Because, even weeks after exposure, PEPs impaired repolarization during stress, and because this trait is pathognomonic of concealed Long QT Syndrome 1 (LQT1) [62, 63] which involves mutations in —or impaired phosphorylative regulation of —Kv7.1, we assessed Kv7.1 serine phosphorylation through immunoprecipitation but found no differences at 10 weeks post-exposure (Additional file 1: Figure S11). This evidence concerns the gene KCNQ1 and familial long QT syndrome.