Furthermore, involvement of the Alcohol-Associated Risk vs. Resilience Network (network 2) differed based on neurodevelopmental outcome—alcohol-exposed children who were typically developing (A/TD) showed activation of MIP-1β, MDC, and MCP-4, and inhibition of CRP and PlGF, with the opposing pattern of cytokine activation/inhibition detected in alcohol-exposed children with neurodevelopmental delay (A/ND), suggesting that activity of this network may play a role in child neurodevelopmental risk vs. resilience. Here, CCL13 is linked to Neurodevelopmental delay.