In the current study, the immune profile of these at-risk children (A/ND) could be disentangled from that of their neurotypical counterparts (A/TD) by analyzing the activity of Alcohol-Associated Risk vs. Resilience Network (network 2): CRP and PlGF levels were elevated and MIP-1β, MDC, and MCP-4 were inhibited in alcohol-exposed children with neurodevelopmental delay, with the opposing profile detected in children with typical neurodevelopment. Here, CCL13 is linked to Neurodevelopmental delay.