Cytoskeletal/contractile proteins followed a similar trend being Filamin‐A (FLNA), Moesin (MSN), Embryonic myosin‐3 (MYH3), Perinatal myosin‐8 (MYH8), Non‐muscle myosin‐9 (MYH9), Embryonic muscle myosin light chain 4 (MYL4), Non‐muscle myosin light polypeptide 6 (MYL6), Spectrin beta chain (SPTB), Tubulin alpha‐1A chain (TUBA1A), Vimentin (VIM), and Alpha actin (ACTC1) increased in DMD compared either with BMD and controls. This evidence concerns the gene TUBA1A and Duchenne muscular dystrophy.