If mOVA2′s capacity for eliciting SIINFEKL-specific CD8 T cell responses is due to infection of-, epitope expression in-, and costimulatory activation of DCs, then adoptive transfer of wt C57Bl6 mice (which do not support poliovirus infection) with ex vivo mOVA2-infected syngeneic FLT3L BMDCs from hCD155-tg mice should have a similar effect. This evidence concerns the gene CD8A and poliovirus infection.