Although genes such as MYH7 (Vermeer et al., 2013), α‐tropomyosin (TPM1) (Kelle, Bentley, Rohena, Cabalka, & Olson, 2016; Nijak et al., 2018), and NKX2.5 (Benson et al., 1999) have been implicated in both EA and LVNC, little is understood regarding the genetic etiology of this disease phenotype. This evidence concerns the gene MYH7 and left ventricular noncompaction.