TP53 and glioblastoma: The presence of M237I p53 amyloid oligomers in glioblastoma cells (Figure 1) and the nuclear M237I p53 oligomers captured in live cells (Figures 3 and 4) indicate that mutant p53 destabilized conformations are present in cells and that, by evading the protein control apparatus, these aberrant p53 forms undergo oligomerization.