Additionally, neither the mRNA nor the protein levels of CS, FAT/CD36, or LPL as well as FAS, SREBP-1c and GPAT1 changed significantly in subjects with multivessel coronary artery disease (Figure 3B, Figure 4B, Figure 5A,C,D, Figure 6A and Figure 7A–C). This evidence concerns the gene GPAM and coronary artery disorder.