Moreover, miR-26a acts on the inflammatory process by downregulating HMGA1 and MALT1, thus impacting the TNF-α/NF-κB inflammatory genes in human bronchial epithelial cells [88]; in addition, the miR-26a/HMGA1 axis controls coronary microembolization-induced myocardial inflammation [87]. Here, HMGA1 is linked to myocarditis.