Besides our studies, anti-Tg glycosylation in HT donors was analyzed by Yuan and coworkers, who showed higher content of sialic acids, mannose, core fucose and Gal(β1,4)GlcNAc(β1,2)Man structures on anti-Tg from HT as compared to anti-Tg from healthy donors. The gene discussed is TG; the disease is hematocrit.