FAS and metabolic dysfunction-associated steatotic liver disease: From a molecular point of view, several genes involved in the disrupted fatty acid synthesis associated with NAFLD, such as sterol regulatory element-binding protein 1c (SREBP-1c), and a series of downstream target genes, such as stearoyl-CoA desaturase-1 (SCD), acetyl-CoA carboxylase (ACC), and fatty acid synthase (FAS), are upregulated in NAFLD patients [4, 5].