TGFB2 and cancer: To identify the source of FA involved in LD formation, we next used 14C-labeled palmitate and showed that the uptake of palmitate by native cancer cells was stimulated upon TGF-β2 treatment (Fig. 4d) and that TGF-β2 silencing in 6.5/cancer cells could prevent palmitate influx, the latter effect being abrogated by the addition of exogenous TGF-β2 (Fig. 4e).