CD36 and atherosclerosis: In addition, foam cell formation and the development of atherosclerosis are inhibited in animal model of CD36 deletion.16 On the other hand, oxLDL induces the up‐regulation of macrophage CD36 in the level of protein and gene, indicating a positive feedback loop in the cause of foam cell formation.17 Thus, further exploring the mechanisms of CD36 expression will be significant for a novel therapeutic scheme for atherosclerosis.