ABL1 and chronic myelogenous leukemia, BCR-ABL1 positive: It was moreover shown that Wnt/Ca2+/NFAT signaling promotes the survival of BCR-ABL-positive chronic myeloid leukemia cells upon inhibition of BCR-ABL1 and that NFAT inhibition enhanced the susceptibility of BCR-ABL positive ALL cells to imatinib [58].