Because CXCL9 and CXCL10 have well-established roles in recruiting CXCR3+ T cells into peripheral tissues under inflammatory conditions [37–41], and because we did not find a difference in Th1/Th2 in papilloma-draining lymph nodes between K17KO and WT mice (S4B Fig), we tested whether recruitment of T cells into the papilloma and early regression in K17KO mice was mediated by the CXCL9/CXCL10/CXCR3 signaling axis. The gene discussed is CXCL9; the disease is papilloma.