Consistent with our reports, recent study demonstrated that METTL3‐mediated m6A modification promoted liver cancer progression.13 Zhang Z et al reported that downregulation of METTL3 significantly impeded bladder cancer cell proliferation, and invasion in vitro and tumorigenicity in vivo.31 Cai X et al also showed that METTL3 promoted the progression of breast cancer by inhibiting tumour suppressor let‐7g.32 In summary, our study robustly demonstrated that METTL3 was significantly upregulated and functioned as an oncogene in human HB. This evidence concerns the gene METTL3 and breast carcinoma.