FGF2 and Miyoshi myopathy: In patients with active MM, monocytes/macrophages under the signaling generated by plasma cell VEGF and FGF-2 production that bind VEGFR-1 and FGFR-1, -2, and -3 expressed on their surface, undergo a phenotypic and functional adaptation which contributes to building neovessels through vasculogenic mimicry [40,41].