TEK and Miyoshi myopathy: In the BM of MM patients with active disease, the endothelial cells display a typical phenotype characterized by the expression on their cellular surface of receptors (i.e., VEGFR-2, FGFR-3, cMET, and Tie2/Tek), increased expression of the β3-integrin, expression of endoglin, and expression of a water transporter, namely aquaporin 1 [21,22].