CCL19 and infection: Cells were subsequently subjected to transwell migration assays toward CCL19, equally conducted as described for Figure 4A. Treatment of mDCs with UV-inactivated HSV-2 virions at an MOI of 5 slightly reduced mDC transwell migration capability toward CCL19 to 70%, while infection with HSV-2 strongly inhibited mDC chemotaxis to a residual migration capability of only 10%.