In certain solid tumours, hypoxic conditions lead to an up‐regulation of LASP112 by binding of HIF1α to a hypoxia response element in the LASP1 promotor region.25 Although the environment in the BM was shown to be hypoxic,27 we were not able to identify a positive correlation between LASP1 and HIF1α in CML within the microarray data sets. This evidence concerns the gene LASP1 and chronic myelogenous leukemia, BCR-ABL1 positive.