LASP1 is overexpressed in several human tumours and is involved in cell proliferation, migration, tumorigenesis and chemoresistance.10, 11 The protein comprises an N‐terminal LIM domain, two nebulin‐like actin binding repeats, a linker region with two phosphorylation sites at S146 and Y171 and a C‐terminal SH3 domain.12 Furthermore, LASP1 has been identified as member of a six genes signature being highly predictive for CML disease phases, thereby allowing a more precise prediction of relapse after stem cell transplantation than clinical risk factors alone.13 The gene discussed is LASP1; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.