In addition, Zhou L et al [41] showed that miR-27a-3p was abnormally highly expressed in gastric cancer tissues and cell lines, and this could induce cell cycle arrest at G1/S to initiate the miR-27a-3p/BTG2/Ras/MEK/ERK pathway by inducing B-cell translocation gene 2 (BTG2) (pro-apoptotic), thereby promoting gastric cancer cell proliferation and tumor growth in vivo and in vitro. Here, BTG2 is linked to neoplasm.