In addition to that, miR-19b-1 overexpression in triple negative breast cancer cells showed a promising effect on arresting tumor growth and angiogenesis, possibly through the down-regulation of VEGFR2, (Ephrin type-B receptor 2) EphB2, and disabled homolog 2 (Dab2), all of which are involved in the internalization of VEGFR2 and the subsequent angiogenesis signaling transduction pathway [133]. The gene discussed is KDR; the disease is triple-negative breast carcinoma.